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Medical Food Designed to Support and Protect the Mitochondrial Function of

Optic Nerve Cells in Patients with Glaucoma

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Mitochondrial Dysfunction in Eye Disease

Mitochondrial Dysfunction in Eye DiseaseThe mitochondria are like the power plants of our cells. We have several thousand mitochondria in nearly every cell of our bodies, and they produce 90 percent of the energy our bodies need to sustain life and support organ function. When these powerhouse cells work less efficiently, a wide range of multi-systemic problems may occur.

The eye and related tissues require a tremendous amount of energy, so it is no surprise that the eyes are particularly vulnerable when mitochondrial dysfunction impairs energy supply.

Mitochondria have attracted a considerable amount of attention from research scientists specializing in the pathophysiology of eye disease — particularly glaucoma. Researchers are working to understand mitochondrial dysfunction and the eyes.

Mitochondrial Dysfunction and Glaucoma

The key to understanding the relationship between mitochondrial dysfunction and glaucoma may be to recognize how mitochondrial dysfunction affects the retinal ganglion cells, or the cells that link the eye to the brain. The eye takes light and converts it to electrical signals that the brain can understand; the retinal ganglion cells are responsible for sending the electrical signals to the brain via axons.

Mitochondrial dysfunction makes the retinal ganglion cells vulnerable to glaucoma injury because it reduces the amount of energy the cell has to repair itself; the dysfunction also makes the retinal ganglion cells more likely to die. Mitochondrial function declines naturally with age.

During the glaucomatous disease process, there is a window of opportunity to treat the optic nerve cells when they are sick so they do not die. The mitochondria die before the optic nerve cells, so intervening to normalize mitochondrial function may help the nerve cells maintain their function and fight off glaucomatous injury.

Offset Mitochondrial Dysfunction

Reverse eye disease

GlaucoCetin is the first medical food specifically designed to support the health of the optic nerve cells by targeting and protecting the underlying mitochondrial function. GlaucoCetin may help to maintain normal metabolism while focusing on protecting these important nerve cells to support vision.

GlaucoCetin combines a powerful range of ingredients shown to be protective and supportive of the neuron mitochondria. This highly active compound is designed specifically to protect optic nerve cells.

For more information about how GlaucoCetin supports and protects mitochondrial function by allowing nerve cells to maintain their function, please contact Guardion Health Sciences today.

Glaucocetin retinal & neuroprotection medical food
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    The Precursor Compound (Glaucohealth™)* was Proven in Clinical Trials, at World-Renowned New York Eye and Ear Infirmary, to Reverse Mitochondrial Dysfunction in Glaucoma Nerve Cells

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    First Medical Food developed specifically to Reverse Damage to Glaucoma Nerve Cells caused by underlying Mitochondrial Dysfunction

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    Comes in Powder Form for Maximum Absorption

*The Precursor to GlaucoCetin, Glaucohealth™, and all related formulas created by Dr Robert Ritch were acquired by Guardion Health Sciences in the second quarter of 2018 and subsequently renamed GlaucoCetin.

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Order your 30-day supply Today

– GlaucoCetin is the first vision-specific Medical Food designed to support and protect the mitochondrial function of nerve cells in glaucoma patients.

30-Day Supply: Comes in Powder Form. 

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GlaucoCetin® is a Medical Food. In order to process your order, you will need a DAC (Doctor Authorization Code) number from your healthcare provider. If you do not have a DAC number, please call us at (800) 873-5141 Dial 1 so that we may assist you.

Please use ONLY letters (A-Z) and numbers (0-9) in the “DAC” field on checkout. No space or symbols.