GlaucoCetin is the first regulated vision-specific Medical Food designed to support and protect the mitochondrial function of optic nerve cells in patients with glaucoma. It contains highly active ingredients intended to support vision by protecting nerve cells. These ingredients are known to be “neuroprotective.”
Many Years of Research
Specific ingredients in GlaucoCetin have been studied for more than 20 years. Its precursor formula, GlaucoHealth, has been proven, in a double-masked, IRB study, to reverse mitochondrial dysfunction of optic nerve cells. It has been used successfully since 2016.
GlaucoCetin Comes in Powder Form
To ensure the inclusion of the highest quantity of ingredients and maximum absorption, GlaucoCetin is provided in a powder form. Powder forms of nutritional supplementation, when evenly mixed with liquid, can deliver important and highly active ingredients into the body very quickly.
GlaucoCetin is the first clinical nutrition supplement designed to support and protect the mitochondrial function of optic nerve cells. It contains highly active ingredients intended to maintain vision by protecting nerve cells. These ingredients are known to be "neuroprotective."
Neuroprotective Natural Ingredients
GlaucoCetin provides a unique way to preserve the optic nerve cells through supporting and protecting the underlying mitochondrial function.
The mitochondria are like the power plants of our cells. We have several thousand mitochondria in nearly every cell of our bodies, and they produce 90 percent of the energy our bodies need to sustain life and support organ function. When these powerhouse cells work less efficiently, a wide range of multi-systemic problems may occur.
The eye and related tissues require a tremendous amount of energy, so it is no surprise that the eyes are particularly vulnerable when mitochondrial dysfunction impairs energy supply.
Researchers are working to understand the implications of mitochondrial dysfunction on the eyes, and the correlation between mitochondrial disorders and the optical system is becoming clearer.
Mitochondrial Dysfunction and Vision
The key to understanding the relationship between mitochondrial dysfunction and vision may be to recognize how mitochondrial dysfunction affects the retinal ganglion cells, or the cells that link the eye to the brain. The eye takes light and converts it to electrical signals that the brain can understand; the retinal ganglion cells are responsible for sending the electrical signals to the brain via axons. If the retinal ganglion cells die, the result may compromise vision.
Mitochondrial dysfunction makes the retinal ganglion cells vulnerable to injury because it reduces the amount of energy the cell has to repair itself; the dysfunction also makes the retinal ganglion cells more likely to die. Mitochondrial function declines naturally with age.
The mitochondria die before the optic nerve cells, so intervening to normalize mitochondrial function may help the nerve cells maintain their function.
Offset Mitochondrial Dysfunction
GlaucoCetin is the first medical food specifically designed to support the health of the optic nerve cells by targeting and protecting the underlying mitochondrial function. GlaucoCetin may help to maintain normal metabolism while focusing on protecting these important nerve cells to maintain vision.
GlaucoCetin combines a powerful range of ingredients shown to be protective and supportive of the neuron mitochondria. This highly active compound is designed specifically to protect optic nerve cells.
For more information about how GlaucoCetin supports and protects mitochondrial function by allowing nerve cells to maintain their function, please contact
Guardion Health Sciences today.
The macular pigment tissue is critical to visual function and protecting the retina from oxidative damage caused by free radicals. The loss of macular pigment can also affect glare sensitivity, or the loss of vision in very bright lighting. A reduced or unhealthy macular pigment may also make it difficult to see properly under low-contrast or low-light situations, such as driving at night, stepping up on a sidewalk in the shadows, or walking through a dim garage.